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Vitamin C reduces air pollution harmful effects on lungs
Sun Aug 19, 2012 1:21PM GMT
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New study suggests that following a vitamin C-rich diet can protect patients suffering from chronic lung diseases against harmful effects of air pollution.
Researchers at Imperial College in London found that vitamin C, acting as an antioxidant, could have a protective role for lungs.
They observed more than 200 patients admitted to hospital for asthma or chronic obstructive pulmonary disease (COPD). They also measured the severity of air pollution on the days before and after the patients’ admission by assessing the levels of "course particulate matter," which is largely produced through the combustion of fossil fuels.
According to the study published in Epidemiology, patients with low levels of vitamin C were at an increased risk of developing breathing problems on days when outdoor air pollution levels were high.
"The protective effect of vitamin C was still present after excluding smokers and elderly subjects, implying that the effect of this antioxidant was not explained by smoking or age," explained Dr Cristina Canova said.
The results uncovered that each 10 micrograms per cubic meter (mcg/m3) rise in the amount of course particulate matter was associated with a 35 percent increase in the risk of hospital admission for patients with asthma or COPD.
"This study adds to a small but growing body of evidence that the effects of air pollution might be modified by antioxidants," said environmental health scientist at the University of British Columbia in Canada Michael Brauer.
Antioxidants, such as vitamin C, may protect the body from harmful molecules called free radicals, counteracting them before they damage cells and cause heart disease, cancer and even respiratory ailments.
The 35-year-old federal law regulating tap water is so out of date that the water Americans drink can pose what scientists say are serious health risks — and still be legal. Examine whether contaminants in your water supply met two standards: the legal limits established by the Safe Drinking Water Act, and the typically stricter health guidelines. The data was collected by an advocacy organization, the Environmental Working Group, who shared it with The Times.
Health Impact News Editor Comments: A new study done at Rhode Island Hospital continues to confirm that Alzheimer’s Disease has a strong link to brain insulin resistance, or type 3 diabetes. Since the National Institute of Health financed the study, we can probably expect people with Alzheimer’s to now be prescribed even more drugs, the same drugs that are prescribed for type 2 diabetes.
Unfortunately, the pharmaceutical companies have a terrible track record when it comes to diabetes drugs. The drug Actos, a drug that France and Germany pulled from the market a year ago after it was found to cause bladder cancer and tumors in people using it for type 2 diabetes, is a drug still found in the market in the United States. Since the FDA has not pulled it off of the market in the US, hundreds of lawsuits have been filed, including one from a whistle blower claiming the manufacturer “is benefiting in tens of billions of dollars in sales at the expense of the lives of people still taking the drug.” Another diabetes drug,Avandia, was removed from pharmacies because it led to increased heart attacks killing thousands of people.
Given all the evidence regarding insulin resistance, diabetes, and the link to Alzheimer’s, it is not surprising that people are reporting incredible benefits from consuming coconut oil. (See: Coconut Oil and Diabetes and Coconut Oil and Alzheimer’s)
For an excellent round table discussion concerning insulin resistance and Alzheimer’s, including the success of coconut oil and a low-carb high-fat diet, watch the following video with 5 medical doctors, Gary Taubes, and Robb Wolf (Article here):
Rhode Island Hospital researcher Suzanne de la Monte, M.D., has found a link between brain insulin resistance (diabetes) and two other key mediators of neuronal injury that help Alzheimer’s disease (AD) to propagate. The research found that once AD is established, therapeutic efforts must also work to reduce toxin production in the brain. The study, Dysfunctional Pro-Ceramide, ER Stress, and Insulin/IGF Signaling Networks with Progression of Alzheimer’s Disease, is published in the June 22, 2012, supplement of the Journal of Alzheimer’s Disease.
Alzheimer’s disease is one of the most common degenerative dementias, and more than 115 million new cases are projected worldwide in the next 40 years. There is clinical and experimental evidence that treatment with insulin or insulin sensitizer agents can enhance cognitive function and in some circumstances help slow the rate of cognitive decline in AD. Alzheimer’s and other neurodegenerative diseases destroy the brain until the patients finally succumb. In order to effectively halt the process of neurodegeneration, the forces that advance and perpetuate the disease, particularly with regard to the progressive worsening of brain insulin/IGF resistance, must be understood.
“Brain insulin resistance (diabetes) is very much like regular diabetes,” de la Monte said. “Since the underlying problems continue to be just about the same, we believe that the development of new therapies would be applicable for all types of diabetes, including Alzheimer’s disease, which we refer to as Type III diabetes.”